Multiple mtDNA deletions with features of MNGIE.

Vissing, J; Ravn, K; Danielsen, E R; Dunø, M; Wibrand, F; Wevers, R A; Schwartz, M · Neurology · 2002

case_report · Level V

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Abstract

Two sisters developed gastrointestinal malabsorption with pain and unsteady gait due to polyneuropathy at age 15. Both had ophthalmoplegia, neurogenic EMG, and COX-negative muscle fibers. One patient had low muscle complex I-IV activity, multiple mtDNA deletions, and depletion, but no thymidine phosphorylase (TP) or dNT-2 gene mutations. TP activity and brain MRI were normal. The condition resembles mitochondrial neurogastrointestinal encephalomyopathy, except for the absence of leukoencephalopathy, and is likely caused by a nuclear DNA mutation that disrupts intergenomic signaling.

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