Carnitine palmitoyltransferase II deficiency: molecular and biochemical analysis of 32 patients.

Wieser, T; Deschauer, M; Olek, K; Hermann, T; Zierz, S · Neurology · 2003

case_series · Level IV

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Abstract

The authors investigated 32 patients with the muscle form of CPT II deficiency. Total carnitine palmitoyltransferase enzyme system (CPT) activity was normal but abnormally inhibited by malonyl-CoA, palmitoyl-CoA, and the detergents Triton X and Tween 20. Mutation analysis identified three described mutations (S113L, P50H, and F448L) and two novel mutations (M214T and Y479F). Using modeling techniques, a structure could be identified anchoring the protein in the membrane. Only one of the five mutations (Y479F) is located within this region.

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