Steroid-responsive neurologic relapses in a child with a proteolipid protein-1 mutation.

Gorman, M P; Golomb, M R; Walsh, L E; Hobson, G M; Garbern, J Y; Kinkel, R P; Darras, B T; Urion, D K et al. · Neurology · 2007

case_report · Level V

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Abstract

A 10-year-old boy developed corticosteroid-responsive relapsing neurologic signs, including nystagmus and ataxia. MRI revealed multifocal T2 white matter hyperintensities; several were gadolinium-enhancing. CSF contained oligoclonal bands. Although the patient met criteria for multiple sclerosis (MS), the proteolipid protein-1 gene (PLP1) contained a mutation in exon 3B (c.409C>T), predicting a tryptophan-for-arginine substitution. This case raises questions about the role of inflammation in PLP1-related disorders and, conversely, PLP1 mutations in MS.

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