Hypothesis: Helicobacter pylori, urease, mucus, and gastric ulcer.
Level V
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- Record sourced from PubMed, PMID 1967668.
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Abstract
Ammonia, released in the gastric mucosa by the action of Helicobacter pylori urease on transuded plasma urea, curtails the biosynthesis of mucus and/or causes the mucus to be disassembled at the mucosal surface. These changes facilitate colonisation by H pylori and may promote gastric ulcer formation.