Duchenne muscular dystrophy models show their age.

Chamberlain, Jeffrey S · Cell · 2010

basic_science · Level V

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Abstract

The lack of appropriate animal models has hampered efforts to develop therapies for Duchenne muscular dystrophy (DMD). A new mouse model lacking both dystrophin and telomerase (Sacco et al., 2010) closely mimics the pathological progression of human DMD and shows that muscle stem cell activity is a key determinant of disease severity.