Nuclear factor-κB in pancreatitis: Jack-of-all-trades, but which one is more important?
editorial · Level V
Where this comes from
- Record sourced from PubMed, PMID 23164573.
- Also identified by DOI 10.1053/j.gastro.2012.11.016 and PMC identifier 6663071.
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Abstract
See “Deletion of IκBα activates RelA to reduce acute pancreatitis in mice through up-regulation of Spi2A,” by Neuhöfer P, Liang S, Einwächter H, et al, on page 192; and “Activation of nuclear factor-κB in acinar cells increases the severity of pancreatitis in mice,” by Huang H, Liu Y, Daniluk J, et al, on page 202.
Medical subject headings
- Acinar Cells
- I-kappa B Kinase
- I-kappa B Proteins
- NF-kappa B
- Pancreatitis
- Serpins
- Transcription Factor RelA
- alpha 1-Antichymotrypsin