Mitochondria at the membrane provide a route to inflammatory cell death.

Chen, Zezhao; Xu, Daichao · Cell Metab · 2026

basic_science · Level V

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Abstract

In a recent issue of Cell, Wang et al. identify "mitoxyperilysis," a previously unknown lytic cell death pathway where combined innate immune and metabolic stress triggers prolonged mitochondria-plasma membrane contact, causing local oxidative damage and membrane rupture. This mTORC2-regulated process identifies a therapeutic axis for inflammatory diseases and cancer.

Medical subject headings