Retinal arterial occlusion in a child with factor V Leiden and thermolabile methylene tetrahydrofolate reductase mutations.

Talmon, T; Scharf, J; Mayer, E; Lanir, N; Miller, B; Brenner, B · Am J Ophthalmol · 1997

case_report · Level V

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Abstract

To analyze the potential cause of retinal arterial occlusion in a 9-year-old child. Case report. Antithrombin III, protein C, free protein S, activated protein C resistance, and antiphospholipid antibodies in plasma were determined. Determination of factor V R506Q (Leiden mutation), thermolabile methylene tetrahydrofolate reductase by polymerase chain reaction, and restriction enzyme analysis were performed. The patient was found to be heterozygous for factor V R506Q (Leiden mutation) and homozygous for thermolabile methylene tetrahydrofolate reductase. Coexistence of two mild hereditary thrombophilic states may result in severe thrombotic manifestations in young people.

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