The potential role of hepatitis C virus in the pathogenesis of the neurological syndrome in chronic hepatitis C.

Caudai, C; Maimone, D; Almi, P; Annunziata, P; Bastianoni, I; Boggiano, C A; Guazzi, G C; Padula, M et al. · Gut · 1997

case_report · Level V

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Abstract

A 72-year-old man developed chronic sensory neuropathy (CSN) during chronic hepatitis C (HCV) infection. Neurological symptoms began one year after acute HCV hepatitis and slowly worsened over three years. No conventional cause for CSN was found. Circulating antinervous tissue antibodies (including anti-Hu) and inflammatory infiltrates in sural nerve biopsy specimens were absent. However, the presence of anti-HCV antibody and HCV-RNA in cerebrospinal fluid indicated that HCV had reached the intrathecal compartment, suggesting the direct viral involvement in the pathogenesis of CSN.

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